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  1. Articles - Development/Plasticity/Repair: ZPK/DLK and MKK4 Form the Critical Gateway to Axotomy-Induced Motoneuron Death in Neonates
    • Takayuki Itoh,
    • Makoto Horiuchi,
    • Raymond H. Ikeda, Jr,
    • Jie Xu,
    • Peter Bannerman,
    • David Pleasure,
    • Josef M. Penninger,
    • Cathy Tournier,
    • and Aki Itoh
    The Journal of Neuroscience, 6 August 2014, 34(32):10729-10742; doi:10.1523/JNEUROSCI.0539-14.2014
  2. Brief Communications: Disruption of NMDA Receptors in Oligodendroglial Lineage Cells Does Not Alter Their Susceptibility to Experimental Autoimmune Encephalomyelitis or Their Normal Development
    • Fuzheng Guo,
    • Yoshiko Maeda,
    • Emily Mills Ko,
    • Monica Delgado,
    • Makoto Horiuchi,
    • Athena Soulika,
    • Laird Miers,
    • Travis Burns,
    • Takayuki Itoh,
    • Haitao Shen,
    • Eunyoung Lee,
    • Jiho Sohn,
    • and David Pleasure
    The Journal of Neuroscience, 11 January 2012, 32(2):639-645; doi:10.1523/JNEUROSCI.4073-11.2012
  3. Brief Communications: ZPK/DLK, a Mitogen-Activated Protein Kinase Kinase Kinase, Is a Critical Mediator of Programmed Cell Death of Motoneurons
    • Aki Itoh,
    • Makoto Horiuchi,
    • Kouji Wakayama,
    • Jie Xu,
    • Peter Bannerman,
    • David Pleasure,
    • and Takayuki Itoh
    The Journal of Neuroscience, 18 May 2011, 31(20):7223-7228; doi:10.1523/JNEUROSCI.5947-10.2011
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