Refine Your Search
1-10 of 10 results
  1. Research Articles - Neurobiology of Disease: Increased resurgent sodium currents in Nav1.8 contribute to nociceptive sensory neuron hyperexcitability associated with peripheral neuropathies
    • Yucheng Xiao,
    • Cindy Barbosa,
    • Zifan Pei,
    • Wenrui Xie,
    • Judith A. Strong,
    • Jun-Ming Zhang,
    • and Theodore R. Cummins
    The Journal of Neuroscience, 0468-18; published ahead of print January 7, 2019, doi:10.1523/JNEUROSCI.0468-18.2018
  2. Articles - Cellular/Molecular: Tetrodotoxin-Resistant Sodium Channels in Sensory Neurons Generate Slow Resurgent Currents That Are Enhanced by Inflammatory Mediators
    • Zhi-Yong Tan,
    • Andrew D. Piekarz,
    • Birgit T. Priest,
    • Kelly L. Knopp,
    • Jeffrey L. Krajewski,
    • Jeff S. McDermott,
    • Eric S. Nisenbaum,
    • and Theodore R. Cummins
    The Journal of Neuroscience, 21 May 2014, 34(21):7190-7197; doi:10.1523/JNEUROSCI.5011-13.2014
  3. Cellular/Molecular: Calmodulin Binds to the C Terminus of Sodium Channels Nav1.4 and Nav1.6 and Differentially Modulates Their Functional Properties
    • Raimund I. Herzog,
    • Chuanju Liu,
    • Stephen G. Waxman,
    • and Theodore R. Cummins
    The Journal of Neuroscience, 10 September 2003, 23(23):8261-8270; doi:10.1523/JNEUROSCI.23-23-08261.2003
  4. ARTICLE - Cellular/Molecular: Nav1.3 Sodium Channels: Rapid Repriming and Slow Closed-State Inactivation Display Quantitative Differences after Expression in a Mammalian Cell Line and in Spinal Sensory Neurons
    • Theodore R. Cummins,
    • Fabio Aglieco,
    • Mathurkrisnan Renganathan,
    • Raimund I. Herzog,
    • Sulayman D. Dib-Hajj,
    • and Stephen G. Waxman
    The Journal of Neuroscience, 15 August 2001, 21(16):5952-5961; doi:10.1523/JNEUROSCI.21-16-05952.2001
  5. ARTICLE - Cellular/Molecular: Glial-Derived Neurotrophic Factor Upregulates Expression of Functional SNS and NaN Sodium Channels and Their Currents in Axotomized Dorsal Root Ganglion Neurons
    • Theodore R. Cummins,
    • Joel A. Black,
    • Sulayman D. Dib-Hajj,
    • and Stephen G. Waxman
    The Journal of Neuroscience, 1 December 2000, 20(23):8754-8761; doi:10.1523/JNEUROSCI.20-23-08754.2000
  6. ARTICLE - Cellular/Molecular: Changes in Expression of Two Tetrodotoxin-Resistant Sodium Channels and Their Currents in Dorsal Root Ganglion Neurons after Sciatic Nerve Injury But Not Rhizotomy
    • Amanda A. Sleeper,
    • Theodore R. Cummins,
    • Sulayman D. Dib-Hajj,
    • William Hormuzdiar,
    • Lynda Tyrrell,
    • Stephen G. Waxman,
    • and Joel A. Black
    The Journal of Neuroscience, 1 October 2000, 20(19):7279-7289; doi:10.1523/JNEUROSCI.20-19-07279.2000
  7. Rapid Communication: A Novel Persistent Tetrodotoxin-Resistant Sodium Current In SNS-Null And Wild-Type Small Primary Sensory Neurons
    • Theodore R. Cummins,
    • Sulayman D. Dib-Hajj,
    • Joel A. Black,
    • Armen N. Akopian,
    • John N. Wood,
    • and Stephen G. Waxman
    The Journal of Neuroscience, 15 December 1999, 19(24):RC43; doi:10.1523/JNEUROSCI.19-24-j0001.1999
  8. ARTICLE: Activation and Inactivation of the Voltage-Gated Sodium Channel: Role of Segment S5 Revealed by a Novel Hyperkalaemic Periodic Paralysis Mutation
    • Saı̈d Bendahhou,
    • Theodore R. Cummins,
    • Rabi Tawil,
    • Stephen G. Waxman,
    • and Louis J. Ptácek
    The Journal of Neuroscience, 15 June 1999, 19(12):4762-4771; doi:10.1523/JNEUROSCI.19-12-04762.1999
  9. ARTICLE: Slow Closed-State Inactivation: A Novel Mechanism Underlying Ramp Currents in Cells Expressing the hNE/PN1 Sodium Channel
    • Theodore R. Cummins,
    • James R. Howe,
    • and Stephen G. Waxman
    The Journal of Neuroscience, 1 December 1998, 18(23):9607-9619; doi:10.1523/JNEUROSCI.18-23-09607.1998
  10. Articles: Downregulation of Tetrodotoxin-Resistant Sodium Currents and Upregulation of a Rapidly Repriming Tetrodotoxin-Sensitive Sodium Current in Small Spinal Sensory Neurons after Nerve Injury
    • Theodore R. Cummins and
    • Stephen G. Waxman
    The Journal of Neuroscience, 15 May 1997, 17(10):3503-3514; doi:10.1523/JNEUROSCI.17-10-03503.1997
Advertisement